Genetic tests showed that certain patients were predisposed to brain injuries if they took the drugs. That information remained secret.

A study conducted by Harvard-affiliated Brigham and Women’s Hospital revealed that individuals experiencing a broad array of long COVID symptoms are twice as likely to have traces of SARS-CoV-2 proteins in their blood compared to those without symptoms of long COVID. The symptoms frequently associated with long COVID include fatigue, brain fog, muscle and joint pain, back pain, headaches, sleep issues, loss of smell or taste, and gastrointestinal problems.These findings were published in the journal Clinical Microbiology and Infection.

IT WAS GETTING late the night of Sept. 12, and Dr. Julian Bailes had settled in to watch a little football. The renowned neurosurgeon and concussions expert tuned in just as Miami Dolphins quarterback Tua Tagovailoa sustained yet another head injury. There was Tagovailoa, lying on the ground with his arms outstretched and his fingers curled into the fencing response, an involuntary position that occurs after a brain injury. To Bailes, it looked almost like a replay from a previous Thursday night game, Sept. 29, 2022, with Tagovailoa on his back, his fingers splayed in front of his face in another automatic response to a concussion.

An innovative study, published in Nature Communications, reveals the mechanism behind two seemingly contradictory effects of fear memories: the inability to forget yet the difficulty to recall. Led by researchers from Sony Computer Science Laboratories, Inc., ATR Computational Neuroscience Laboratories, and the University of Tokyo, the study shows how fear experiences are initially remembered as broad, associative memories, but over time become integrated into episodic memories with a more specific timeline.

The blast shook the ground and its red flash of fire covered the doorway as U.S. special operations forces blew open a door during a recent training exercise. Moments later, in their next attempt, the boom was noticeably suppressed and the blaze a bit smaller, testament to just one of the new technologies that U.S. Special Operations Command is using to limit the brain injuries that have become a growing problem for the military.

Depression can make it tougher for athletes to recover from a concussion, and vice versa, a new study finds. Student athletes who have both concussion and depression have significantly worse symptoms for both conditions, researchers reported recently in the journal Brain Research. What's more, electrical signals in the brain are more disrupted among people with both concussion and depression than among those with only one of these conditions, the researchers noted.

A California veteran was booted off a Delta Air Lines flight because her T-shirt that featured a message about veteran suicide rates in the U.S. Catherine Banks was supposed to fly out of San Francisco on Wednesday, but instead she was forced off the plane by a flight attendant who found her shirt 'threatening.' The shirt read: 'Do not give in to the war within. End veteran suicide.'

The exact nature of long COVID is still coming to light, but we just got some of the best evidence yet that this debilitating condition stems from a brain injury. Using high-resolution scanners, researchers at the Universities of Cambridge and Oxford have shown microscopic, structural abnormalities in the brainstems of those recovering from COVID-19.

It has been 24 years since a pharmaceutical was last approved for posttraumatic stress disorder (PTSD). The condition is notoriously difficult to treat, with up to 40% patients finding no relief from symptoms through psychotherapy or current medications. Many clinicians, advocates, and patients had pinned their hopes on the psychedelic drug midomafetamine with assisted therapy (MDMA-AT). However, in August, the US Food and Drug Administration (FDA) rejected it. At this point, it’s unclear when the therapy will be available, if ever.

Researchers used mouse models and human post-mortem brain tissue to assess the molecular underpinnings that may increase the risk of Alzheimer’s disease following traumatic brain injuries (TBI). The findings appeared in Acta Neuropathologica.