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Question Are repetitive head impacts during a professional football career associated with mortality among National Football League players?
Findings In this cohort study of 13 912 National Football League players, a 25% increase in repetitive head impacts during a professional football career was associated with a statistically significant increase in the hazard ratio of death.
Meaning The findings suggest that repetitive head impacts are associated with an increase in the risk of all-cause mortality among professional football players.
The national age-adjusted rate of fall-related TBI deaths increased by 17% from 2008 to 2017; rates increased significantly in 29 states and among nearly all groups, most notably persons living in noncore nonmetropolitan counties and those aged ≥75 years.
The association of dispositional optimism with health-related factors has been well established in several clinical populations, but little is known about the role of optimism in recovery after traumatic brain injury (TBI). Given the high prevalence of cognitive complaints after TBI, the present study examined the association between optimism and cognitive functioning after TBI.
Traumatic brain injury (TBI) is a leading cause of death and disability. Older adults are more likely than younger individuals to sustain TBIs and less likely to survive them. TBI has been called the “silent epidemic,” and older adults are the “silent population” within this epidemic. This study evaluates whether indicators of preinjury health and functioning are associated with risk of incident traumatic brain injury (TBI) with loss of consciousness (LOC) and to evaluate health‐related factors associated with mortality in individuals with incident TBI.
Traumatic brain injury (TBI) can have lifelong and dynamic effects on health and wellbeing. Research on the longterm consequences emphasises that, for many patients, TBI should be conceptualised as a chronic health condition. Evidence suggests that functional outcomes after TBI can show improvement or deterioration up to two decades after injury, and rates of all-cause mortality remain elevated for many years. Furthermore, TBI represents a risk factor for a variety of neurological illnesses, including epilepsy, stroke, and neurodegenerative disease. With respect to neurodegeneration after TBI, post-mortem studies on the long-term neuropathology after injury have identified complex persisting and evolving abnormalities best described as polypathology, which includes chronic traumatic encephalopathy. Despite growing awareness of the lifelong consequences of TBI, substantial gaps in research exist. Improvements are therefore needed in understanding chronic pathologies and their implications for survivors of TBI, which could inform long-term health management in this sizeable patient population.
Concussion is a common childhood injury that may lead to long-term physical, behavioral, and neurocognitive effects, affecting learning and school performance. There is increasing concern about the potential for repeat concussions among professional and high school athletes, with specific attention focused on understanding how sustaining a concussion alters future concussion risk. Addressing repeat concussion risk among youth has substantial implications for clinical practice in terms of managing exposure — particularly regarding youth sports participation — and long-term health and development.
The American Academy of Pediatrics (AAP) has updated its concussion recommendations to support children and teens engaging in light physical activity and returning to school as they recover. The report, revised for the first time in eight years, also advises against complete removal of electronic devices following a concussion.
Light-induced pain or discomfort, or ‘photalgia’ (otherwise known as light sensitivity, photophobia, asthenopia, photoallodynia or photodynia) is a well-known consequence of mild traumatic brain injury.
“You’re supposed to rest after concussion.” This advice has been given countless times to patients recovering from concussion. How did we get here, what is the evidence that brought us here, and what does the evolving evidence actually tell us?
A Harvard Medical School study published in Nature Communications shows a molecular relationship between TBI and CTE but also a possible treatment mechanism — just not one that can be used to treat humans quite yet.