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Explosions and Blast Injuries: A Primer for Clinicians
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As the risk of terrorist attacks increases in the US, disaster response personnel must understand the unique pathophysiology of injuries associated with explosions and must be prepared to assess and treat the people injured by them.
Key Concepts
- Bombs and explosions can cause unique patterns of injury seldom seen outside combat.
- The predominant post explosion injuries among survivors involve standard penetrating and blunt trauma. Blast lung is the most common fatal injury among initial survivors.
- Explosions in confined spaces (mines, buildings, or large vehicles) and/or structural collapse are associated with greater morbidity and mortality.
- Half of all initial casualties will seek medical care over a one-hour period. This can be useful to predict demand for care and resource needs.
- Expect an “upside-down” triage — the most severely injured arrive after the less injured, who bypass EMS triage and go directly to the closest hospitals.
Background
Explosions can produce unique patterns of injury seldom seen outside combat. When they do occur, they have the potential to inflict multi-system life-threatening injuries on many persons simultaneously. The injury patterns following such events are a product of the composition and amount of the materials involved, the surrounding environment, delivery method (if a bomb), the distance between the victim and the blast, and any intervening protective barriers or environmental hazards. Because explosions are relatively infrequent, blast-related injuries can present unique triage, diagnostic, and management challenges to providers of emergency care.
Few U.S. health professionals have experience with explosive-related injuries. Vietnam-era physicians are retiring, other armed conflicts have been short-lived, and until this past decade, the U.S. was largely spared of the scourge of mega-terrorist attacks. This primer introduces information relevant to the care of casualties from explosives and blast injuries.
Classification of Explosives
Explosives are categorized as high-order explosives (HE) or low-order explosives (LE). HE produce a defining supersonic over-pressurization shock wave. Examples of HE include TNT, C-4, Semtex, nitroglycerin, dynamite, and ammonium nitrate fuel oil (ANFO). LE create a subsonic explosion and lack HE’s over-pressurization wave. Examples of LE include pipe bombs, gunpowder, and most pure petroleum-based bombs such as Molotov cocktails or aircraft improvised as guided missiles. HE and LE cause different injury patterns.
Explosive and incendiary (fire) bombs are further characterized based on their source. “Manufactured” implies standard military-issued, mass produced, and quality-tested weapons. “Improvised” describes weapons produced in small quantities, or use of a device outside its intended purpose, such as converting a commercial aircraft into a guided missile. Manufactured (military) explosive weapons are exclusively HE-based. Terrorists will use whatever is available – illegally obtained manufactured weapons or improvised explosive devices (also known as “IEDs”) that may be composed of HE, LE, or both. Manufactured and improvised bombs cause markedly different injuries.
Blast Injuries
The four basic mechanisms of blast injury are termed as primary, secondary, tertiary, and quaternary (Table 1). “Blast Wave” (primary) refers to the intense over-pressurization impulse created by a detonated HE. Blast injuries are characterized by anatomical and physiological changes from the direct or reflective over-pressurization force impacting the body’s surface. The HE “blast wave” (over-pressure component) should be distinguished from “blast wind” (forced super-heated air flow). The latter may be encountered with both HE and LE.
LE are classified differently because they lack the self-defining HE over-pressurization wave. LE’s mechanisms of injuries are characterized as due from ballistics (fragmentation), blast wind (not blast wave), and thermal. There is some overlap between LE descriptive mechanisms and HE’s Secondary, Tertiary, and Quaternary mechanisms.
This Explosives Primer was developed from published and unpublished sources. If quoted, please cite date and time as changes will be made as new information becomes available or is cleared for public distribution. From the U.S. Department of Health and Human Services and the Centers for Disease Control and Prevention, 2006.







